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Publications

Innate Immune Memory to Repeated Borrelia burgdorferi Exposure Correlates with Murine In Vivo Inflammatory Phenotypes

by MAO | Oct 27, 2025 | Publications

Innate Immune Memory to Repeated Borrelia burgdorferi Exposure Correlates with Murine In Vivo Inflammatory Phenotypes…

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Protective Immunity and New Vaccines for Lyme Disease

by MAO | Oct 27, 2025 | Publications

Protective Immunity and New Vaccines for Lyme Disease Discovery of the mechanism of action of the first vaccine…

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Blood Smears Have Poor Sensitivity for Confirming Borrelia miyamotoi Disease

by MAO | Oct 27, 2025 | Publications

Blood Smears Have Poor Sensitivity for Confirming Borrelia miyamotoi DiseaseBorrelia miyamotoi disease (BMD) is a…

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Early Questing by Lone Star Tick Larvae, New York and Massachusetts, USA, 2018

by MAO | Oct 27, 2025 | Publications

Early Questing by Lone Star Tick Larvae, New York and Massachusetts, USA, 2018 Subtropical lone star tick larvae…

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Strain-specific joint invasion and colonization by Lyme disease spirochetes is promoted by outer surface protein C

by MAO | Oct 27, 2025 | Publications

Strain-specific joint invasion and colonization by Lyme disease spirochetes is promoted by outer surface protein C The…

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The intergenic small non-coding RNA ittA is required for optimal infectivity and tissue tropism in Borrelia burgdorferi

by MAO | Oct 27, 2025 | Publications

The intergenic small non-coding RNA ittA is required for optimal infectivity and tissue tropism in Borrelia…

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The Tufts University Lyme Disease Initiative is a group of clinicians and scientists committed to working together to end Lyme disease as a threat to human health.  Taking a multi-pronged approach and partnering with researchers at other universities, in government and in industry, we are exploring unique strategies to meet our goal by the year 2030.  Visit our research pages to learn about our approaches to Lyme disease.

Recent Posts

Comparative reservoir competence of Peromyscus leucopus, C57BL/6J, and C3H/HeN for Borrelia burgdorferi B31

Borrelia burgdorferi initiates early transcriptional re-programming in macrophages that supports long-term suppression of inflammation

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